Acute fatty liver of pregnancy (AFLP) has been found
to be associated in some cases with an abnormality of fatty-acid metabolism. This
abnormality is a deficiency of the enzyme long-chain-3-hydroxyacyl-CoA dehydrogenease
(LCHAD).
T he mother (and father) have 50% of normal LCHAD activity and the fetus has
no LCHAD activity.
The metabolic disease in the baby's liver apparently causes the fatty liver disease
in the mother. In cases of AFLP due to LCHAD deficiency, there is a 25% (or greater)
risk of AFLP in each pregnancy.
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